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Pioglitazone acutely reduces insulin secretion and causes metabolic deceleration of the pancreatic beta-cell at submaximal glucose concentrations


By JPGRAY - Posted on 23 August 2009

TitlePioglitazone acutely reduces insulin secretion and causes metabolic deceleration of the pancreatic beta-cell at submaximal glucose concentrations
Publication TypeJournal Article
Year of Publication2009
AuthorsLamontagne J, Pepin E, Peyot ML, Joly E, Ruderman NB, Poitout V, Madiraju SR, Nolan CJ, Prentki M
JournalEndocrinology
Volume150
Issue8
Pagination3465-74
Date PublishedAug
Publication Languageeng
ISBN Number1945-7170 (Electronic)
Accession Number19406947
Key WordsAdenosine Triphosphate/metabolism, Animals, Berberine/pharmacology, Cell Line, Fatty Acids/metabolism, Glucose/*pharmacology, Hypoglycemic Agents/*pharmacology, Immunoblotting, Insulin/*secretion, Insulin-Secreting Cells/*drug effects/*metabolism, Islets of Langerhans/drug effects/metabolism, Lipid Metabolism/drug effects, Membrane Potential, Mitochondrial/drug effects, Metformin/pharmacology, Pyrazoles/pharmacology, Pyrimidines/pharmacology, Rats, Rats, Wistar, Thiazolidinediones/*pharmacology
Abstract

Thiazolidinediones (TZDs) have beneficial effects on glucose homeostasis via enhancement of insulin sensitivity and preservation of beta-cell function. How TZDs preserve beta-cells is uncertain, but it might involve direct effects via both peroxisome proliferator-activated receptor-gamma-dependent and -independent pathways. To gain insight into the independent pathway(s), we assessed the effects of short-term (

Notes

DK019514-26/DK/NIDDK NIH HHS/United StatesIn VitroJournal ArticleResearch Support, N.I.H., ExtramuralResearch Support, Non-U.S. Gov'tUnited States

URLhttp://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Citation&list_uids=19406947
Citation Key567
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